Dr. Nawab John Dar receives prestigious recognition from Marquie Who is Who in US

In a remarkable testament to his scientific prowess, Dr. Nawab John Dar has received prestigious recognition from Marquie Who is Who in America for his contributions to the field of neuroscience.

It is intended as an accurate source of biographical information that can be referenced by subscription by “librarians, students, researchers, corporate executives, journalists, personnel recruiters and many others,” according to Marquis Who’s Who.

Those included are selected because their “accomplishments were found to be of significant reference value.” With a steadfast commitment to unraveling the intricacies of the human brain, Dr. Dar’s work has significantly advanced our understanding of neurodegenerative disorders, with a particular focus on Alzheimer’s Disease (AD).

Having graduated from CSIR-Indian Institute of Integrative Medicine, Jammu, India, Dr. Dar embarked on a research journey focused on elucidating the role of Withanolides in paradigms of Glutamate/NMDA excitotoxicity. Through his meticulous investigations, Dr. Dar made significant strides in comprehending the neuroprotective potential of distinct Withanolides, effectively protecting neuronal cells from oxidative stress and various toxic insults.

Continuing his academic pursuit, Dr. Dar pursued a transformative Postdoctoral Fellowship at St. Boniface Hospital/University of Manitoba, Canada, under the guidance of Dr. Gordon Glazner.

Here, his research was dedicated to unraveling the intricate mechanisms underlying neurogenesis, with a specific focus on the role of secreted amyloid precursor protein alpha. Dr. Dar’s findings shed light on the interplay between molecular signaling and brain regeneration, offering new perspectives on the fundamental processes governing brain development.

In 2019, Dr. Dar boarded on a new chapter in his scientific odyssey, joining the University of Texas Health Sciences Center, San Antonio. There, he embarked on a pioneering investigation into the role of glutathione peroxidase 4 (GPX4) and the phenomenon of Oxytosis/ferroptosis in mouse models of Alzheimer’s Disease. Through his meticulous research, Dr. Dar unveiled the detrimental consequences of GPX4 downregulation and subsequent lipid peroxidation, uncovering functional deficits in neural stem cell differentiation.

Driven by an unyielding desire to unravel the complexities of neurodegenerative disorders, Dr. Dar joined the distinguished Salk Institute in May 2022, in Professor Pamela Maher’s group. Together, they embarked on a groundbreaking endeavor to investigate the role of oxytosis/ferroptosis in Alzheimer’s disease. Their current research endeavors focus on the development of an advanced in vitro model of chronic toxicity. By meticulously examining the molecular mechanisms involved over an extended period, Dr. Dar is aiming to unravel the progressive nature of neurodegeneration. Ultimately, their goal is to identify specific therapeutic targets that could pave the way for effective interventions against this devastating condition.

At the Salk Institute, Dr. Dar thrives in an environment that fosters scientific excellence. He acknowledges the invaluable mentorship of his supervisor and the exceptional support of his colleagues, emphasizing that collaborative efforts are key to groundbreaking scientific advancements. Dr. Dar considers himself fortunate to be part of an institution that cultivates an unwavering commitment to scientific inquiry, making his scientific journey immensely fulfilling.

Through his esteemed recognition in Marquie Who is Who, Dr. Dar’s contributions to the field of neuroscience receive well-deserved acclaim. His tireless dedication to unraveling the complexities of neurodegenerative disorders brings us closer to unlocking novel therapeutic strategies. With unwavering passion and a profound scientific acumen, Dr. Dar continues to pave the way for groundbreaking discoveries that hold the potential to transform the lives of individuals affected by neurodegenerative diseases.